It functions by tying to the GLP-1 receptor on pancreatic cells, another GPCR. The intestine releases GLP-1 after a meal, which causes the pancreas to release insulin. Cells are then prompted to absorb glucose from the blood by insulin.
- Both the GLP-1 and glucagon receptors are class B GPCRs. Several class A GPCRs have had their structures determined, but due to technical difficulties, class B receptors haven't received as much attention. On June 8, 2017, four worldwide research teams published their findings in Nature regarding the glucagon and GLP-1 receptor architecture. NIH's National Institute on Drug Abuse (NIDA), National Institute of General Medical Sciences (NIGMS), and National Institute on Diabetes and Digestive and Kidney Diseases (NIDDK) provided some funding to two of the teams (NIDA).
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